This is a collection of studies that was compiled by another researcher. I first came upon the list in 2015 when I began studying the issue. I read most of them and they led me deeper into the study of vaccines.
While there is no one study that conclusively proves that vaccines cause autism, that is because that is not how science works. We still have to think and puzzle and put the pieces together ourselves. These studies should help the curious, open minded person get started. 1 http://www.ncbi.nlm.nih.gov/pubmed/21058170 giving the Hepatitis B vaccine to newborn baby boys could triple the risk of developing an autism spectrum disorder 2 http://omsj.org/reports/tomljenovic%202011.pdf Aluminum, a highly neurotoxic metal and the most commonly used vaccine adjuvant may be a significant contributing factor to the rising prevalence of ASD 3 http://www.ncbi.nlm.nih.gov/pubmed/21623535 Researchers found a positive and statistically significant relationship between autism and vaccinations. 4 http://www.hindawi.com/journals/jt/2013/801517/ They determined that ASD patients have a heightened sensitivity to thimerosal which would restrict cell proliferation that is typically found after vaccination. 5 http://www.ncbi.nlm.nih.gov/pubmed/12145534 The study concludes that the autistic children had an inappropriate or abnormal antibody response to MMR. 6 http://ajcn.nutrition.org/content/80/6/1611.full An increased vulnerability to oxidative stress and a decreased capacity for methylation may contribute to the development and clinical manifestation of autism. 7 http://www.ncbi.nlm.nih.gov/pubmed/11895129 Recent studies indicate that transition metals act as catalysts in the oxidative reactions of biological macromolecules therefore the toxicities associated with these metals might be due to oxidative tissue damage. 8 http://www.ncbi.nlm.nih.gov/pubmed/14745455 The potent inhibition of this pathway by ethanol, lead, mercury, aluminum and thimerosal suggests that it may be an important target of neuro developmental toxins. 9 https://imfar.confex.com/imfar/2010/webprogram/Paper5280.html Our studies demonstrate that the level of MS mRNA in the brain is high during fetal development and gradually decreases with age. This decrease is likely to be important for the normal progression of development, via its influence over DNA and histone methylation. MS mRNA levels in the cortex are abnormally low in autism, which is likely to reflect the presence of oxidative stress. Therapies which counteract oxidative stress and promote methylation may be a useful strategy in treating autism. 10 http://jcn.sagepub.com/content/22/11/1308.abstract a significant relation does exist between the blood levels of mercury and diagnosis of an autism spectrum disorder. 11 http://www.ncbi.nlm.nih.gov/pubmed/15527868 Thimerosal-induced cytotoxicity was associated with depletion of intracellular GSH in both cell lines. 12 http://journals.plos.org/plosone/article?id=10.1371/journal.pone.0068444 Elevation in peripheral oxidative stress is consistent with, and may contribute to, the more severe functional impairments in the ASD-GID group. 13 http://www.ncbi.nlm.nih.gov/pubmed/16338635 On average, for each 1,000 lb of environmentally released mercury, there was a 43% increase in the rate of special education services and a 61% increase in the rate of autism. 14 http://www.ncbi.nlm.nih.gov/pubmed/12933322 Hair excretion patterns among autistic infants were significantly reduced relative to control. These data cast doubt on the efficacy of traditional hair analysis as a measure of total mercury exposure in a subset of the population. 15 http://www.ncbi.nlm.nih.gov/pubmed/17454560 There was a significant dose-response relationship between the severity of the regressive ASDs observed and the total mercury dose children received from Thimerosal-containing vaccines. 16 http://civileats.com/wp-content/uploads/2009/01/palmer2008.pdf We found that for every 1000 pounds of industrial release, there was a corresponding 2.6% increase in autism rates (po.05) and a 3.7% increase associated with power plant emissions (Po.05). Distances to these sources were independent predictors after adjustment for relevant covariates. For every 10 miles from industrial or power plant sources, there was an associated decreased autism Incident Risk of 2.0% and 1.4%, respectively (po.05). 17 http://www.ane.pl/pdf/7020.pdf maturational changes in amygdala volume and the binding capacity of [11C]DPN in the amygdala was significantly altered in infant macaques receiving the vaccine schedule. 18 http://www.ncbi.nlm.nih.gov/pubmed/18482737 Consistent significantly increased rate ratios were observed for autism, autism spectrum disorders, tics, attention deficit disorder, and emotional disturbances with Hg exposure from Thimerosal containing vaccines. 19 http://www.ncbi.nlm.nih.gov/pubmed/19357975 As a result of the present findings, in combination with the brain pathology observed in patients diagnosed with autism, the present study helps to support the possible biological plausibility for how low-dose exposure to mercury from thimerosal-containing vaccines may be associated with autism. 20 http://labmed.ascpjournals.org/content/33/9/708.full.pdf Depressed immunity, autoimmunity, and inflammatory activation are common features in autism. Impaired resistance to infection may predispose to chronic measles infection of the autistic gut by MMR vaccine. Thimerosal-containing vaccine during infancy may depress immunity and lower the threshold for chronic vaccinial measles infection. 21 http://www.ncbi.nlm.nih.gov/pmc/articles/PMC3264864/?tool=pubmed neonatal exposure to thimerosal-containing vaccines might induce excitotoxic brain injuries, leading to neurodevelopmental disorders. 22 http://mercury-freedrugs.org/docs/00mmdd_EISAbstractSubmission_IncreasedRiskOfDevelopmentalNeurologicImpairmentAfterHighExposureToThimerosal-containingVaccine_.pdf This analysis suggests that high exposure to ethyl mercury from thimerosal containing vaccines in the first month of life increases the risk of subsequent development of neurologic development impairment 23 http://www.hindawi.com/journals/jt/2014/491316/ Humans are increasingly exposed to Al from food, water, medicinals, vaccines, and cosmetics, as well as from industrial occupational exposure. Al disrupts biological self-ordering, energy transduction, and signaling systems, thus increasing biosemiotic entropy. 24 http://www.ncbi.nlm.nih.gov/pubmed/16126512 Vaccines, in several reports were found to be temporally followed by a new onset of autoimmune diseases. The same mechanisms that act in infectious invasion of the host, apply equally to the host response to vaccination. It has been accepted for diphtheria and tetanus toxoid, polio and measles vaccines and GBS. Also this theory has been accepted for MMR vaccination and development of autoimmune thrombocytopenia, MS has been associated with HBV vaccination. 25 http://www.ms.academicjournals.org/article/article1409245960_Deisher%20et%20al.pdf Change points in these countries corresponded to introduction of or increased doses of human fetal cell line manufactured vaccines, while no relationship was found between paternal age or Diagnostic and Statistical Manual (DSM) revisions and autistic disorder diagnosis. Further, linear regression revealed that Varicella and Hepatitis A immunization coverage was significantly correlated to autistic disorder cases. R software was used to calculate change points. Autistic disorder change points years are coincident with introduction of vaccines manufactured using human fetal cell lines, containing fetal and retroviral contaminants, into childhood vaccine regimens. This pattern was repeated in the US, UK, Western Australia and Denmark. Thus, rising autistic disorder prevalence is directly related to vaccines manufactured utilizing human fetal cells. 26 http://www.ncbi.nlm.nih.gov/pubmed/19747466?itool=EntrezSystem2.PEntrez.Pubmed.Pubmed_ResultsPanel.Pubmed_RVDocSum&ordinalpos=1 Pharmacokinetic analysis revealed that Hg from THIM injections accumulates in the rat brain in significant amounts and remains there longer than 30 days after the injection. At the 6th week of age animals were examined for pain sensitivity using the hot plate test. THIM treated rats of both strains and sexes manifested statistically significantly elevated pain threshold (latency for paw licking, jumping) on a hot plate (56 degrees C). 27 http://www.termedia.pl/Original-paper-Lasting-neuropathological-changes-in-rat-brain-after-intermittent-neonatal-administration-of-thimerosal,20,15811,1,1.html administration of THIM to suckling rats in a vaccination-like manner and at doses analogous to those used in paediatric vaccines or higher injures neurons and astroglia in several brain regions. 28 http://www.ncbi.nlm.nih.gov/pubmed/21549155 early postnatal THIM administration causes lasting neurobehavioral impairments and neurochemical alterations in the brain, dependent on dose and sex. If similar changes occur in THIM/mercurial-exposed children, they could contribute do neurodevelopmental disorders. 29 http://www.ncbi.nlm.nih.gov/pubmed/23843785 certain individuals with a mild mitochondrial defect may be highly susceptible to mitochondrial specific toxins like the vaccine preservative thimerosal. 30 http://www.ncbi.nlm.nih.gov/pmc/articles/PMC3395253/ ethylmercury not only inhibits mitochondrial respiration leading to a drop in the steady state membrane potential, but also concurrent with these phenomena increases the formation of superoxide, hydrogen peroxide, and Fenton/Haber-Weiss generated hydroxyl radical. These oxidants increase the levels of cellular aldehyde/ketones. Additionally, we find a five-fold increase in the levels of oxidant damaged mitochondrial DNA bases and increases in the levels of mtDNA nicks and blunt-ended breaks. 31 http://www.ncbi.nlm.nih.gov/pubmed/25433158 Our study demonstrated that serum TRX levels were associated with ASD, and elevated levels could be considered as a novel, independent diagnosis indicator of ASD. 32 http://www.ncbi.nlm.nih.gov/pubmed/18321861 Overall, mercury inhibition was selective toward the thioredoxin system. In particular, the remarkable potency of the mercury compounds to bind to the selenol-thiol in the active site of TrxR should be a major molecular mechanism of mercury toxicity. 33 http://www.ncbi.nlm.nih.gov/pubmed/20810785 These results stress the role of TrxR as a target of mercurials and provide the mechanism of selenite as a detoxification agent for mercury poisoning. 34 http://www.ncbi.nlm.nih.gov/pubmed/9756729 This study is the first to report an association between virus serology and brain autoantibody in autism; it supports the hypothesis that a virus-induced autoimmune response may play a causal role in autism. 35 http://www.sciencedirect.com/science/article/pii/S0041008X06001335 Coproporphyrin levels were elevated in children with autistic disorder relative to control groups. Elevation was maintained on normalization for age or to a control heme pathway metabolite (uroporphyrin) in the same samples. The elevation was significant (P < 0.001). Porphyrin levels were unchanged in Asperger's disorder, distinguishing it from autistic disorder. The atypical molecule precoproporphyrin, a specific indicator of heavy metal toxicity, was also elevated in autistic disorder (P < 0.001) but not significantly in Asperger's. A subgroup with autistic disorder was treated with oral dimercaptosuccinic acid (DMSA) with a view to heavy metal removal. Following DMSA there was a significant (P = 0.002) drop in urinary porphyrin excretion. These data implicate environmental toxicity in childhood autistic disorder. 36 http://archpsyc.jamanetwork.com/article.aspx?articleid=208897 This study validates the existence of early autistic regression. 37 http://www.mdpi.com/1099-4300/14/11/2227 Our results provide strong evidence supporting a link between autism and the aluminum in vaccines. A literature review showing toxicity of aluminum in human physiology offers further support. Mentions of autism in VAERS increased steadily at the end of the last century, during a period when mercury was being phased out, while aluminum adjuvant burden was being increased. Using standard log-likelihood ratio techniques, we identify several signs and symptoms that are significantly more prevalent in vaccine reports after 2000, including cellulitis, seizure, depression, fatigue, pain and death, which are also significantly associated with aluminum-containing vaccines. We propose that children with the autism diagnosis are especially vulnerable to toxic metals such as aluminum and mercury due to insufficient serum sulfate and glutathione. A strong correlation between autism and the MMR (Measles, Mumps, Rubella) vaccine is also observed, which may be partially explained via an increased sensitivity to acetaminophen administered to control fever. 38 http://jcn.sagepub.com/content/21/2/170.abstract These data suggest that further metabolic evaluation is indicated in autistic patients and that defects of oxidative phosphorylation might be prevalent. Children who have mitochondrial related dysfunctional cellular energy, metabolism might be more prone to undergo autistic regression between 18 and 30 months of age if they also have infections or immunizations at the same time. 39 http://www.ncbi.nlm.nih.gov/pubmed/16151044 The most replicated finding in autism neuroanatomy-a tendency to unusually large brains-has seemed paradoxical in relation to the specificity of the abnormalities in three behavioral domains that define autism. We now know a range of things about this phenomenon, including that brains in autism have a growth spurt shortly after birth and then slow in growth a few short years afterward, that only younger but not older brains are larger in autism than in controls, that white matter contributes disproportionately to this volume increase and in a nonuniform pattern suggesting postnatal pathology, that functional connectivity among regions of autistic brains is diminished, and that neuroinflammation (including microgliosis and astrogliosis) appears to be present in autistic brain tissue from childhood through adulthood. Alongside these pervasive brain tissue and functional abnormalities, there have arisen theories of pervasive or widespread neural information processing or signal coordination abnormalities (such as weak central coherence, impaired complex processing, and underconnectivity), which are argued to underlie the specific observable behavioral features of autism. This convergence of findings and models suggests that a systems- and chronic disease-based reformulation of function and pathophysiology in autism needs to be considered, and it opens the possibility for new treatment targets. Oxidative stress, brain inflammation and microgliosis have been much documented in association with toxic exposures including various heavy metals...the awareness that the brain as well as medical conditions of children with autism maybe conditioned by chronic biomedical abnormalities such as inflammation opens the possibility that meaningful biomedical interventions maybe possible well past the window of maximal neuroplasticity in early childhood because the basis for assuming that all deficits can be attributed to fixed early developmental alterations in neural architecture has now been undermined. 40 http://www.ncbi.nlm.nih.gov/pubmed/17090484 This article discusses the evidence for the case that some children with autism may become autistic from neuronal cell death or brain damage sometime after birth as result of insult; and addresses the hypotheses that toxicity and oxidative stress may be a cause of neuronal insult in autism. 41 http://www.ncbi.nlm.nih.gov/pubmed/16766163 There is a positive correlation between reduced levels of these proteins and loss of previously acquired language skills in children with autism. Taken together, these studies suggest increased oxidative stress in autism that may contribute to the development of this disease. A mechanism linking oxidative stress with membrane lipid abnormalities, inflammation, aberrant immune response, impaired energy metabolism and excitotoxicity, leading to clinical symptoms and pathogenesis of autism is proposed. 42 http://www.ncbi.nlm.nih.gov/pubmed/17114826 Behavioral testing showed motor deficits in the aluminum treatment group that expressed as a progressive decrease in strength measured by the wire-mesh hang test (final deficit at 24 wk; about 50%). Significant cognitive deficits in water-maze learning were observed in the combined aluminum and squalene group (4.3 errors per trial) compared with the controls (0.2 errors per trial) after 20 wk. Apoptotic neurons were identified in aluminum-injected animals that showed significantly increased activated caspase-3 labeling in lumbar spinal cord (255%) and primary motor cortex (192%) compared with the controls. Aluminum-treated groups also showed significant motor neuron loss (35%) and increased numbers of astrocytes (350%) in the lumbar spinal cord. The findings suggest a possible role for the aluminum adjuvant in some neurological features associated with GWI and possibly an additional role for the combination of adjuvants. 43 http://montanaim.com/pubs/Heavy_Metals_Article.pdf Metals are ubiquitous in our environment, and exposure to them is inevitable. However, not all people accumulate toxic levels of metals or exhibit symptoms of metal toxicity, suggesting that genetics play a role in their potential to damage health. Metal toxicity creates multisystem dysfunction, which appears to be mediated primarily through mitochondrial damage from glutathione depletion. 44 http://www.icdrc.org/documents/Mitoandautism2008.pdf Exposure to environmental toxins is the likely etiology for MtD in autism. 45 http://www.tandfonline.com/doi/abs/10.1080/02772240701806501#.VOJrTS5DGZ9 The odds of receiving education intervention services were approximately nine times as great for vaccinated boys (n = 46) as for unvaccinated boys (n = 7), after adjustment for confounders. 46 http://www.jneuroinflammation.com/content/pdf/1742-2094-7-20.pdf HgCl2 stimulates VEGF and IL-6 release from human mast cells. This phenomenon could disrupt the blood-brain-barrier and permit brain inflammation. As a result, the findings of the present study provide a biological mechanism for how low levels of mercury may contribute to ASD pathogenesis. 47 http://www.genome-explorations.com/images/pdfs_publications/10-Cultured%20lymphocytes%20from%20autistic%20children%20and%20non-autis.pdf Cultured lymphocytes challenged with zinc responded with an impressive up-regulation of MT transcripts (at least nine different MTs were over-expressed) while cells challenged with thimerosal responded by up-regulating numerous heat shock protein transcripts, but not MTs. 48 http://www.vaccines.net/vaccine-induced-immune-overload.pdf There has been an epidemic of inflammatory diseases that has paralleled the epidemic on iatrogenic immune stimulation with vaccines. The epidemic of diabetes/prediabetes appears to be accelerating at a time when the prevalence of obesity has stabilized, indicating that the negative feedback system of the immune system has been over whelmed. The theory of vaccine induced immune overload explains the key observations that have confounded many competing hypothesis. Unfortunately the prospective controlled trials of vaccines performed for licensure are either too small, too short in duration or inappropriately controlled (use other vaccines as controls) to appropriately study the relationship between vaccines and these epidemics. Furthermore most epidemiological studies performed after licensure of vaccines suffer from the same deficiencies. 49 http://www.ncbi.nlm.nih.gov/pubmed/20576582 These findings identify disordered porphyrin metabolism as a salient characteristic of autism. 50 http://www.nature.com/mp/journal/v17/n3/full/mp2010136a.html Mitochondrial dysfunction in autism spectrum disorders: a systematic review and meta-analysis. 51 http://www.sciencedirect.com/science/article/pii/S0300483X10002040 Sensitization effect of thimerosal is mediated in vitro via reactive oxygen species and calcium signaling. 52 http://informahealthcare.com/doi/abs/10.3109/1547691X.2010.545086 Theoretical aspects of autism: Causes—A review Documented causes of autism include genetic mutations and/or deletions, viral infections, and encephalitis following vaccination. 53 http://www.ncbi.nlm.nih.gov/pubmed/21797771 The results showed the prevalence rate of ASD among the grandchildren of pink disease survivors (1 in 22) to be significantly higher than the comparable general population prevalence rate (1 in 160). The results support the hypothesis that Hg sensitivity may be a heritable/genetic risk factor for ASD. 54 http://www.ncbi.nlm.nih.gov/pubmed?term=Risk%20Factors%20for%20Autistic%20Regression%3A%20Results%20of%20an%20Ambispective%20Cohort%20Study. This study suggests that febrile seizures and family history of neuropsychiatric disorders are correlated with autistic regression. 55 http://www.ncbi.nlm.nih.gov/pubmed/15265850 MMR vaccination was associated with a transient increased rate of febrile seizures 56 http://journals.plos.org/plosone/article?id=10.1371/journal.pone.0027897 Adverse Events following 12 and 18 Month Vaccinations: a Population-Based, Self-Controlled Case Series Analysis 57 http://www.ncbi.nlm.nih.gov/pmc/articles/PMC2957583/ These data document that exposure to thimerosal during early postnatal life produces lasting alterations in the densities of brain opioid receptors along with other neuropathological changes, which may disturb brain development. 58 http://www.ebcala.org/unanswered-questions In 21 published cases of the Court of Federal Claims, which administers the VICP, the Court stated that the petitioners had autism or described autism unambiguously. This finding calls into question the decisions of the Court of Federal Claims in the Omnibus Autism Proceeding in 2009 and 2010 and the statement of Health and Human Services on its website that “HHS has never concluded in any case that autism was caused by vaccination.” 59 http://www.ncbi.nlm.nih.gov/pubmed/21350943 Thimerosal at concentrations relevant for infants' exposure (in vaccines) is toxic to cultured human-brain cells and to laboratory animals. the neurotoxic effect of ethylmercury has not been studied with co-occurring adjuvant-Al in thimerosal containing vaccines. 60 http://www.ncbi.nlm.nih.gov/pubmed/22249285 We conclude that exposure of Hepa1-6 cells to a low dose of adjuvanted hepatitis B vaccine leads to loss of mitochondrial integrity, apoptosis induction, and cell death, apoptosis effect was observed also in C2C12 mouse myoblast cell line after treated with low dose of vaccine (0.3, 0.1, 0.05 μg/ml). In addition In vivo apoptotic effect of hepatitis B vaccine was observed in mouse liver. 61 http://toxsci.oxfordjournals.org/content/92/1/246.full.pdf+html Taken together, these results indicate that thimerosal-induced neurotoxicity occurs through the JNK-signaling pathway, independent of cJun activation, leading ultimately to apoptotic cell death. 62 http://www.ncbi.nlm.nih.gov/pubmed/22015705 TM exposure resulted in a delayed startle response in SD neonates and decreased motor learning in SHR male (22.6%), in SD male (29.8%), and in SD female (55.0%) neonates. TM exposure also resulted in a significant increase in cerebellar levels of the oxidative stress marker 3-nitrotyrosine in SHR female (35.1%) and SD male (14.0%) neonates. The activity of cerebellar type 2 deiodinase, responsible for local intra-brain conversion of thyroxine to the active hormone, 3',3,5-triiodothyronine (T3), was significantly decreased in TM-exposed SHR male (60.9%) pups. This coincided with an increased (47.0%) expression of a gene negatively regulated by T3, Odf4 suggesting local intracerebellar T3 deficiency. Our data thus demonstrate a negative neurodevelopmental impact of perinatal TM exposure which appears to be both strain- and sex-dependent. 63 http://www.ncbi.nlm.nih.gov/pubmed/19234401 Autism incidence in California shows no sign yet of plateauing. Younger ages at diagnosis, differential migration, changes in diagnostic criteria, and inclusion of milder cases do not fully explain the observed increases. Other artifacts have yet to be quantified, and as a result, the extent to which the continued rise represents a true increase in the occurrence of autism remains unclear. 64 http://www.ncbi.nlm.nih.gov/pmc/articles/PMC3204610/#!po=5.55556 However, further research is needed to confirm that the mild inflammatory response elicited by vaccination is benign in pregnancy. 65 http://www.ncbi.nlm.nih.gov/pubmed/23337946 This finding suggests that maternal inflammation may have a significant role in autism 66 http://www.ncbi.nlm.nih.gov/pmc/articles/PMC3364648/#!po=2.00000 What is regressive autism and why does it occur? Is it the consequence of multi-systemic dysfunction affecting the elimination of heavy metals and the ability to regulate neural temperature? 67 http://www.ncbi.nlm.nih.gov/pubmed/23867105 Immunological and autoimmune considerations of Autism Spectrum Disorders. 68 http://journals.plos.org/plosone/article?id=10.1371/journal.pone.0058058 Identification of Unique Gene Expression Profile in Children with Regressive Autism Spectrum Disorder (ASD) and Ileocolitis 69 http://www.ncbi.nlm.nih.gov/pubmed/6182806 The results indicate the existence of a cell-mediated immune response to brain tissue in the syndrome of autism. 70 http://www.ncbi.nlm.nih.gov/pubmed/10759242 Controls were all negative. The sequences obtained from the patients with Crohn's disease shared the characteristics with wild-strain virus. The sequences obtained from the patients with ulcerative colitis and children with autism were consistent with being vaccine strains. The results were concordant with the exposure history of the patients. Persistence of measles virus was confirmed in PBMC in some patients with chronic intestinal inflammation. 71 http://lup.sagepub.com/content/21/2/118.full The spectrum of ASIA: ‘Autoimmune (Auto-inflammatory) Syndrome induced by Adjuvants’ 72 http://www.ncbi.nlm.nih.gov/pubmed/18197631 Thiol-modulated mechanisms of the cytotoxicity of thimerosal and inhibition of DNA topoisomerase II alpha. 73 http://www.nature.com/nature/journal/v501/n7465/full/nature12504.html Here we find that topotecan, a topoisomerase 1 (TOP1) inhibitor, dose-dependently reduces the expression of extremely long genes in mouse and human neurons, including nearly all genes that are longer than 200 kilobases... many high-confidence ASD candidate genes are exceptionally long and were reduced in expression after TOP1 inhibition. Our findings suggest that chemicals and genetic mutations that impair topoisomerases could commonly contribute to ASD and other neurodevelopmental disorders. 74 http://link.springer.com/article/10.1007%2Fs12026-013-8403-1#page-1 We have examined the neurotoxicity of aluminum in humans and animals under various conditions, following different routes of administration, and provide an overview of the various associated disease states. The literature demonstrates clearly negative impacts of aluminum on the nervous system across the age span. In adults, aluminum exposure can lead to apparently age-related neurological deficits resembling Alzheimer’s and has been linked to this disease and to the Guamanian variant, ALS–PDC. Similar outcomes have been found in animal models. In addition, injection of aluminum adjuvants in an attempt to model Gulf War syndrome and associated neurological deficits leads to an ALS phenotype in young male mice. In young children, a highly significant correlation exists between the number of pediatric aluminum-adjuvanted vaccines administered and the rate of autism spectrum disorders. Many of the features of aluminum-induced neurotoxicity may arise, in part, from autoimmune reactions, as part of the ASIA syndrome. 75 http://app.autism360.org/MumperPrevention.pdf Emerging research suggests that the timing of environmental factors in the presence of genetic predispositions has influenced the increase in autism spectrum disorders over the past several decades. A review of the medical literature suggests that autism may be impacted by environmental toxicants, breastfeeding duration, gut flora composition, nutritional status, acetaminophen use, vaccine practices and use of antibiotics and/or frequency of infections. The author reports her retrospective clinical research in a general pediatric practice (Advocates for Children), which shows a modesttrend toward lower prevalence of autism than her previous pediatric practice or recent CDC data. Out of 294 general pediatrics patients followed since 2005 there were zero new cases of autism (p value 0.014). Given the prevalence of autism for that cohort of 1 in 50 children in the United States, it is important to consider implementing strategies in primary care practice that could potentially modify environmental factors or affect the timing of environmental triggers contributing to autism. 76 http://www.ncbi.nlm.nih.gov/pubmed/10589903 Measles inclusion-body encephalitis caused by the vaccine strain of measles virus. 77 http://www.ncbi.nlm.nih.gov/pmc/articles/PMC3930734/ Difficulties in Eliminating Measles and Controlling Rubella and Mumps: A Cross-Sectional Study of a First Measles and Rubella Vaccination and a Second Measles, Mumps, and Rubella Vaccination.
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Doctors who explain clearly why vaccines aren't safe or effective. This is an old list. Many of these resources have been removed from the internet. I leave them here as a historical document.
1. Dr. Nancy Banks - http://bit.ly/1Ip0aIm 2. Dr. Russell Blaylock - http://bit.ly/1BXxQZL 3. Dr. Shiv Chopra - http://bit.ly/1gdgh1s 4. Dr. Sherri Tenpenny - http://bit.ly/1MPVbjx 5. Dr. Suzanne Humphries - http://bit.ly/17sKDbf 6. Dr. Larry Palevsky - http://bit.ly/1LLEjf6 7. Dr. Toni Bark - http://bit.ly/1CYM9RB 8. Dr. Andrew Wakefield - http://bit.ly/1MuyNzo 9. Dr. Meryl Nass - http://bit.ly/1DGzJsc 10. Dr. Raymond Obomsawin - http://bit.ly/1G9ZXYl 11. Dr. Ghislaine Lanctot - http://bit.ly/1MrVeUL 12. Dr. Robert Rowen - http://bit.ly/1SIELeF 13. Dr. David Ayoub - http://bit.ly/1SIELve 14. Dr. Boyd Haley PhD - http://bit.ly/1KsdVby 15. Dr. Rashid Buttar - http://bit.ly/1gWOkL6 16. Dr. Roby Mitchell - http://bit.ly/1gdgEZU 17. Dr. Ken Stoller - http://bit.ly/1MPVqLI 18. Dr. Mayer Eisenstein - http://bit.ly/1LLEqHH 19. Dr. Frank Engley, PhD - http://bit.ly/1OHbLDI 20. Dr. David Davis - http://bit.ly/1gdgJwo 21. Dr Tetyana Obukhanych - http://bit.ly/16Z7k6J 22. Dr. Harold E Buttram - http://bit.ly/1Kru6Df 23. Dr. Kelly Brogan - http://bit.ly/1D31pfQ 24. Dr. RC Tent - http://bit.ly/1MPVwmu 25. Dr. Rebecca Carley - http://bit.ly/K49F4d 26. Dr. Andrew Moulden - http://bit.ly/1fwzKJu 27. Dr. Jack Wolfson - http://bit.ly/1wtPHRA 28. Dr. Michael Elice - http://bit.ly/1KsdpKA 29. Dr. Terry Wahls - http://bit.ly/1gWOBhd 30. Dr. Stephanie Seneff - http://bit.ly/1OtWxAY 31. Dr. Paul Thomas - http://bit.ly/1DpeXPf 32. Many doctors talking at once - http://bit.ly/1MPVHOv 33. Dr. Richard Moskowitz - censored 34. Dr. Jane Orient - http://bit.ly/1MXX7pb 35. Dr. Richard Deth - http://bit.ly/1GQDL10 36. Dr. Lucija Tomljenovic - http://bit.ly/1eqiPr5 37. Dr Chris Shaw - http://bit.ly/1IlGiBp 38. Dr. Susan McCreadie - http://bit.ly/1CqqN83 39. Dr. Mary Ann Block - http://bit.ly/1OHcyUX 40. Dr. David Brownstein - http://bit.ly/1EaHl9A 41. Dr. Jayne Donegan - http://bit.ly/1wOk4Zz 42. Dr. Troy Ross - censored 43. Dr. Philip Incao - http://bit.ly/1ghE7sS 44. Dr. Joseph Mercola - http://bit.ly/18dE38I 45. Dr. Jeff Bradstreet - http://bit.ly/1MaX0cC 46. Dr. Robert Mendelson - http://bit.ly/1JpAEQr 47. Dr Theresa Deisher https://m.youtube.com/watch?feature=youtu.be&v=6Bc6WX33SuE 48. Dr. Sam Eggertsen https://m.youtube.com/watch?v=8LB-3xkeDAE Hundreds more doctors testifying that vaccines aren't safe or effective, in these documentaries 1. Vaccination - The Silent Epidemic - http://bit.ly/1vvQJ2W 2. The Greater Good - http://bit.ly/1icxh8j 3. Shots In The Dark - http://bit.ly/1ObtC8h 4. Vaccination The Hidden Truth - http://bit.ly/KEYDUh 5. Vaccine Nation - http://bit.ly/1iKNvpU 6. Vaccination - The Truth About Vaccines - http://bit.ly/1vlpwvU 7. Lethal Injection - http://bit.ly/1URN7BJ 8. Bought - http://bit.ly/1M7YSlr 9. Deadly Immunity - http://bit.ly/1KUg64Z 10. Autism - Made in the USA - http://bit.ly/1J8WQN5 11. Beyond Treason - http://bit.ly/1B7kmvt 12. Trace Amounts - http://bit.ly/1vAH3Hv 13. Why We Don't Vaccinate - http://bit.ly/1KbXhuf 14. Autism Yesterday - (2010) - http://bit.ly/1URU2A7 9 hour court case https://m.youtube.com/watch?v=DFTsd042M3o |
AuthorAll posts are by Elliott Freed, author of Vaccine Primer, unless otherwise noted. ArchivesCategories |